New molecular mechanism in diastolic heart failure.
نویسندگان
چکیده
In contrast to systolic heart failure (SHF), for which knowledge of pathophysiology and therapy has advanced rapidly over the past decade, little is known about diastolic heart failure (DHF). The article by van Heerebeek et al1 in this issue of Circulation that describes an abnormal distribution of titin isoforms in DHF may herald a new approach to understanding the pathophysiology of this syndrome.
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ورودعنوان ژورنال:
- Circulation
دوره 113 16 شماره
صفحات -
تاریخ انتشار 2006